Answer to your Health Question
Dynamic left ventricular outflow tract (LVOT) obstruction is thought to be a hallmark of hypertrophic obstructive cardiomyopathy, especially in those cases with isolated asymmetric septal hypertrophy and systolic anterior motion (SAM) of the mitral valve.
Recently, several authors described the occurrence of a dynamic LVOT obstruction during acute coronary insufficiency in ventricles without significant myocardial hypertrophy.
LVOT obstruction in the setting of acute myocardial infarction could predispose to cardiac rupture.
The development of a LVOT obstruction during acute anterior myocardial infarction has to be considered a serious and potentially fatal complication.
Transesophageal echocardiography (TEE) provides unique diagnostic capabilities, allowing for a very precise look at the structure and hemodynamics of the human heart.
It is minimally invasive and portable, and quickly diagnoses sudden hemodynamic changes in intensive care patients.
It provides invaluable and precise information about myocardial dysfunction and intracardiac volume status.
It can diagnose dynamic left ventricular outflow obstruction.
left ventricular outflow tract obstruction is not an uncommon phenomenon,
but it is often unrecognized. Its rapid recognition and effective treatment
is only possible with transesophageal echocardiography (TEE).
The Doppler effect is a phenomenon observed whenever the source of waves is moving with respect to an observer.
Doppler interrogation of the left ventricular outflow tract provides direct documentation and quantization of outflow tract obstruction.
Excessive volume extraction and sympathetic activation may be the possible causes of Dynamic left ventricular outflow track obstruction in a patient with end-stage renal failure on haemodialysis.
Systolic anterior motion of the mitral valve and mitral-septal contact is the usual cause of dynamic left ventricular outflow obstruction in hypertrophic cardiomyopathy.
Although left ventricular outflow tract obstruction is commonly associated with congenitally corrected transposition of the great vessels, this obstruction is seldom caused by accessory mitral valve tissue.
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